Tachycardia (Rapid arrhythmias)

Arrhythmias are abnormal heart rhythms that may lead to severe complications such as heart failure, stroke, or sudden cardiac arrest. Our electrophysiologists team specializes in managing tachycardia (rapid arrhythmias), utilizing advanced diagnostics and tailored therapies to improve outcomes.

Atrial Fibrillation (AF): Atrial fibrillation is a chaotic, irregular heartbeat originating in the atria, significantly increasing risks of stroke, systemic embolism, and heart failure. Uncontrolled AF can impair cardiac output and accelerate structural heart damage, demanding prompt intervention to mitigate life-threatening consequences.

Atrial Flutter (AFL): Atrial flutter involves rapid, organized atrial rhythms often exceeding 250 bpm. While less chaotic than AF, it still poses high thromboembolic risks and may rapidly decompensate cardiac function, particularly in post-surgical cases where scarring complicates electrical pathways.

Supraventricular Tachycardia (SVT)：SVT arises from aberrant circuits above the ventricles, provoking sudden palpitations, dizziness, or syncope. Though rarely fatal, recurrent episodes severely impair quality of life and may mask underlying cardiac pathology.

Premature Atrial Contractions (PACs)：PACs are extra heartbeats starting in the atria, often harmless with symptoms like palpitations.While often benign, high-burden APCs correlate with atrial remodeling and possibility of progression to sustained arrhythmias like AF or AFL.

Atrial Tachycardia (AT): Focused on abnormal atrial foci, AT causes sustained rapid heart rates, potentially triggering cardiomyopathy or hemodynamic instability. Persistent episodes may exacerbate underlying heart failure and require urgent rhythm control.

Premature Ventricular Contractions (PVCs): PVCs always arise from ventricular foci.High-density PVCs may induce ventricular dysfunction or degenerate into ventricular tachycardia. Risk stratification is vital, as malignant forms signal underlying ischemia or cardiomyopathy requiring aggressive management.

Ventricular Tachycardia (VT): VT is a life-threatening rhythm originating in the ventricles, associated with sudden cardiac death, especially in structural heart disease. Immediate intervention is critical to prevent collapse or fatal arrhythmic storms.

Vasovagal Syncope (VVS): Triggered by exaggerated reflex responses, this condition causes abrupt drops in heart rate and blood pressure, leading to transient loss of consciousness. Recurrent episodes raise risks of traumatic injury and necessitate personalized preventive strategies.

Bradycardia

Severe bradycardia, particularly in cases of sick sinus syndrome and second-degree or higher atrioventricular blocks, commonly manifests as a constellation of clinical symptoms including fatigue, dizziness, palpitations, exertional intolerance, transient vision loss (amaurosis fugax), and syncopal episodes. This cardiac rhythm disturbance can be effectively managed through minimally invasive pacemaker implantation, which restores appropriate heart rate and improves hemodynamic stability.

Prevetion of sudden cardiac death

Implantable Cardioverter Defibrillator (ICD) therapy constitutes a critical intervention for high-risk populations susceptible to sudden cardiac death, particularly in managing life-threatening ventricular arrhythmias associated with structural heart diseases (e.g., heart failure, cardiomyopathies) and inherited arrhythmia syndromes (e.g., long QT syndrome, Brugada syndrome). This advanced cardiac rhythm management system provides both continuous rhythm monitoring and immediate defibrillation capabilities, which has demonstrated significant mortality reduction in these vulnerable patient cohorts.

Heart failure associated with cardiac asynchrony

Cardiac resynchronization therapy (CRT) represents an advanced biventricular pacing modality that restores coordinated ventricular contraction through precisely timed electrical stimulation. This evidence-based therapeutic intervention is particularly indicated for patients with systolic heart failure (LVEF ≤35%) accompanied by electrical dyssynchrony (QRS duration ≥130ms), including those with ischemic or non-ischemic cardiomyopathy. The procedure demonstrates significant clinical efficacy in improving hemodynamic parameters, reducing mitral regurgitation, and reversing ventricular remodeling when implemented in appropriately selected candidates.